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p smad2 3  (MedChemExpress)


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    MedChemExpress p smad2 3
    P Smad2 3, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/p+smad2+3/pm42045912-143-41-43?v=MedChemExpress
    Average 94 stars, based on 1 article reviews
    p smad2 3 - by Bioz Stars, 2026-07
    94/100 stars

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    Immunohistochemical analysis of β-catenin, p <t>-SMAD2/3,</t> and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).
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    Immunohistochemical analysis of β-catenin, p <t>-SMAD2/3,</t> and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).
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    Immunohistochemical analysis of β-catenin, p <t>-SMAD2/3,</t> and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).
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    Immunohistochemical analysis of β-catenin, p <t>-SMAD2/3,</t> and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).
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    Immunohistochemical analysis of β-catenin, p <t>-SMAD2/3,</t> and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).
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    Immunohistochemical analysis of β-catenin, p -SMAD2/3, and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).

    Journal: Regenerative Therapy

    Article Title: Role of WIF-1 in regulating chondrocyte dedifferentiation and matrix production through WNT pathway

    doi: 10.1016/j.reth.2026.101083

    Figure Lengend Snippet: Immunohistochemical analysis of β-catenin, p -SMAD2/3, and p -JNK in early-passage, late-passage, WIF1-silenced, and WIF1-overexpressing human auricular chondrocytes. (a–c) IHC staining of β-catenin, p -SMAD2/3, and p -JNK in early-passage (P2) and late-passage (P7) chondrocyte pellets. (d–f) IHC staining of the same markers in P2 chondrocytes transduced with scramble shRNA or shWIF1, and in P7 pellets transduced with Empty control or WIF1 overexpression lentivirus. (e, g, i) High-magnification views corresponding to β-catenin, p -SMAD2/3, and p -JNK staining in control versus WIF1-modulated groups. Scale bars: 100 μm (low magnification), 20 μm (high magnification).

    Article Snippet: p -SMAD2/3 , Bioss , bs-8853R , Polyclonal , 1:200 , IHC.

    Techniques: Immunohistochemical staining, Immunohistochemistry, Transduction, shRNA, Control, Over Expression, Staining